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    <title>eCommons Community: Cornell University Library</title>
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    <title>Herd outbreak of bovine respiratory syncytial virus (BRSV) : clinically affected cows due to a lack of biosecurity?</title>
    <link>http://hdl.handle.net/1813/11449</link>
    <description>Title: Herd outbreak of bovine respiratory syncytial virus (BRSV) : clinically affected cows due to a lack of biosecurity?
&lt;br/&gt;
&lt;br/&gt;Authors: Marvin, Jennifer A.</description>
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    <title>Generalized Tetanus in a Ten-week-old Puppy</title>
    <link>http://hdl.handle.net/1813/11448</link>
    <description>Title: Generalized Tetanus in a Ten-week-old Puppy
&lt;br/&gt;
&lt;br/&gt;Authors: Marini, Lauren
&lt;br/&gt;
&lt;br/&gt;Abstract: Generalized limb stiffness and an inability to eat were seen as the presenting complaints in a ten-week-old puppy. The puppy had been castrated and received vaccinations approximately twelve days before developing a left hindlimb lameness, which progressed to extreme stiffness of all four limbs. On presentation, the dog was also noted to be hyperthermic with contraction of facial muscles causing the lips to be pulled back and the ears to be seated atop the head. These findings strongly suggested a diagnosis of generalized tetanus, for which the dog was treated and recovered fully in approximately two months. This report describes the etiology and pathogenesis of C. tetani infection. It explores both the generalized and focal forms of the disease and will also discuss treatment.</description>
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    <title>Surgical repair of a cleft soft palate in a 3 month old Fell Pony Foal; a case review</title>
    <link>http://hdl.handle.net/1813/11447</link>
    <description>Title: Surgical repair of a cleft soft palate in a 3 month old Fell Pony Foal; a case review
&lt;br/&gt;
&lt;br/&gt;Authors: MacNaughton, Darren
&lt;br/&gt;
&lt;br/&gt;Abstract: A three month old Fell Pony filly presented for definitive diagnosis and surgical correction of a congenitally cleft soft palate. Since birth she had copious amounts of mucoid nasal discharge bilaterally, hypersalivation, and after nursing, milk was visible from both nostrils. An endoscopic examination of her oral and nasal cavities revealed a large and wide, central, soft palate defect extending the entire length of the palate. Thoracic radiographs showed an increased diffuse airspace pattern in her cranioventral lungfields consistent with pneumonia. Bloodwork revealed a mature neutrophilic leukocytosis, with no left shift or toxic changes noted.&#xD;
	The soft palate defect was surgically corrected using a combination of two techniques; a laryngotomy and a lip sparing mandibular symphysiotomy, in an effort to get maximum exposure and repair of the defect. Immediately after surgery she began to nurse showing little evidence of milk regurgitation, but she retained a mild serous nasal discharge. Her surgical pain was managed via transdermal fentanyl patch, and her aspiration pneumonia was treated with a course of chloramphenicol. Post-operatively she continued to have a mild serous nasal discharge and occasional regurgitant nasal reflux. Her aspiration pneumonia resolved and she continued to do well despite these persistent problems. She recently required a second surgery to repair a one inch dehiscence of the previous repair in the caudal most aspect of her soft palate. Congenitally cleft soft palates in horses are rare and pose a serious challenge to correct surgically due to lack of adequate exposure, and high complication rate.</description>
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    <title>Review of Etiologic Hypotheses for Feline Hyperthyroidism</title>
    <link>http://hdl.handle.net/1813/11446</link>
    <description>Title: Review of Etiologic Hypotheses for Feline Hyperthyroidism
&lt;br/&gt;
&lt;br/&gt;Authors: Lynn, Amy
&lt;br/&gt;
&lt;br/&gt;Abstract: Feline hyperthyroidism (FH) was first reported in 1979 and is now recognized as the most common feline endocrinopathy. The pathogenesis of the disease remains unclear despite numerous epidemiological and molecular studies reported since that first case. Whether FH is a new disease, an epidemic of a rare disease, or can be attributed to an older cat population, increased veterinarian and owner awareness, or improved diagnostic testing is not known. Several risk factors for FH have been identified but none has proven to be both necessary and sufficient to cause the disease. The cause of FH is likely mulltifactorial.</description>
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